Treatment options for acute myocardial infarction

1. The formation of myocardial infarction

Myocardial infarction is due to insufficient coronary blood supply to the heart, resulting in partial myocardial necrosis due to severe ischemia. The following are several key factors in the development of myocardial infarction:

1.1 Coronary artery occlusion

Coronary atherosclerosis can lead to narrowing or occlusion of the coronary artery lumen, which is the main cause of myocardial infarction. Atherosclerotic plaque rupture or thrombus formation in coronary arteries can lead to acute coronary artery blockage, leading to myocardial ischemia and necrosis.

1.2 Myocardial ischemia and necrosis

Coronary artery occlusion leads to myocardial ischemia, in which cardiomyocytes die due to hypoxia and ischemia. This necrotic process is irreversible, and damaged cardiomyocytes cannot regenerate.

1.3 Release of cardiac enzymes

When heart muscle cells die due to ischemia, enzymes and other substances within the cells are released into the blood. These substances can be used as diagnostic markers of myocardial infarction, such as CK-MB (creatine kinase isoenzyme).

2. Symptoms of Myocardial Infarction

Symptoms of a heart attack may include the following:

2.1 Angina pectoris

Angina pectoris is a typical symptom of myocardial infarction. It usually manifests as a sense of oppression, chest tightness, and chest pain, which can radiate to the back, left shoulder, and left upper arm. Angina may last for a long time and the pain may gradually increase.

2.2 Arrhythmia

Myocardial infarction may cause cardiac arrhythmias, including fast or slow heartbeat, atrial fibrillation, ventricular tachycardia, and others. Arrhythmia may affect the heart's ability to pump blood, leading to heart failure.

2.3 Cardiogenic shock

Severe myocardial infarction may lead to cardiogenic shock, manifested as low blood pressure, rapid heartbeat, clammy skin, and confusion. Cardiogenic shock can be life-threatening if left untreated.

2.4 Heart failure

Myocardial necrosis caused by myocardial infarction will affect the pumping function of the heart, and patients may experience symptoms of heart failure, such as dyspnea, lung rales, and fatigue.

So how to prevent the sudden onset of acute myocardial infarction! To solve the problem fundamentally is to repair one's own metabolism

So how to go to metabolic repair, what is metabolic repair?

Metabolic repair, derived from the transformation of the Nobel Prize-winning theory, this technology mobilizes the ubiquitin-proteasome system to fully metabolize and decompose pathogenic proteins, and at the same time guides the huge energy generated by metabolism to release the potential of cell self-replication, and finally through the cell The way of self-replication completes the self-repair of tissues and organs, so that the system function returns to normal and the body regains real health. Metabolic repair is a life science technology that stimulates the body's self-repair ability to fight against various lifestyle diseases. Hippocrates, the father of medicine, asserted as early as the third century BC, "'natural force' is an effective way to cure diseases!" The "natural force" he mentioned is the ability to repair itself.

 

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